Joaquín Morales

ADA - Animal Data Analytics - Spain Author

He was born in Barcelona in 1973 and graduated from the Faculty of Veterinary Medicine of the Autonomous University of Barcelona in 1996. Afterwards he obtained his PhD in Veterinary Medicine in 2002 from the aforementioned University, in the Animal Nutrition and Feeding Department and under the direction of Dr. José Francisco Pérez and Dr. Mariola Baucells. His PhD thesis dealt with the digestive and metabolic differences for the use of different carbohydrates sources between two types of clearly differentiated genetics: the Landrace, as the representative of the white pig, and the Iberian pig.

Since then, he started to work in the R&D Deparrtment of ADA - Animal Data Analytics (PigCHAMP Pro Europa), a company located in Segovia whose activity is directed at the animal (mainly swine) production consultancy.

Currently, Joaquín is the person in charge of this R&D Department, and he is responsible for the development of projects that are financed with competitive public funds and for the experimental tests with contracts with companies of the swine sector. His main duties belong to the experimental design, the control of the development of the tests, the data analysis, the final report and, if it comes to that, the divulgation of the results in different national and international journals. As a result of these studies and of the development of his PhD thesis more than 15 international publications and more than 50 conferences in congresses have come up.

Updated CV 09-Mar-2013

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27-May-2020maria.rodriguezDear Alan
Thank you very much for your comment.
In this trial we used 34 sows in batch 1 (9 primiparous and 23 multiparous) and 35 sows in batch 2 (4 primiparous and 31 multiparous).
Regards
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13-Apr-2017FrancisExactly what are the key cytokines involved in the gut immunostimulation?
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26-Mar-2014mester.karoly_1The question is not so simple. Increase the fermentable fiber is a good idea, but it does not resolve the issue.
The sow's head two interesting symptom. The eye conjunctiva inflammation. The sow mouth on the edge of white foam. In which I am not sure, but I see the picture of the sow snout skin tissue necrosis.
This causes additional problems. I think that the sow does not compensate, combo, acidosis, metabolitikus and respiratory system eye visible symptom. The other big problem with the release of ammonia poisoning, a symptom of the frothing at the mouth.
To compensate for the acidosis enters the renal route, this is known as escaping the glutamine → 2 HCO3- + 2 NH+ 4. The kidneys of the ammonium ions should choose, but I do not know. At this point, the Organization would reach the goal of having a glutamine residue, namely the two breaks down to HCO3-ion which would compensate the acidosis. Further it is possible to process the liver detoxication function where the urea cycle in the detoxication of ammonia. The liver does not go in the urea cycle,
Two reasons
1. small number of intact liver parenchyma cells in the liver.
2. lysine-arginine antagonism of the urea cycle, unable to function.
This causes the ammonia poisoning. Due to the unfortunate sow liver and kidney damage.
The acidosis can develop kidney problems, liver problems due to ketosis is the sow it causes a huge acid load the animal. The respiratory acidosis due to kidney failure can develop.
Renal insufficiency → blood pressure increase → hypertrophic heart in work → blood stasis in the pulmonary circulation → pulmonary oedema → reduced the effectiveness of respiration, carbon dioxide exhalation → respiratory acidosis.
Ammonia poisoning symptom:
Ammonia is added to the liquor, the displacement of the ammonia paralyses muscle motor cells. Therefore, you can not swallow the sow, so it builds up in the saliva foam in the mouth. The nervous excitement of ammonia poisoning is caused by the State. Muscle tremors, gnash one’s teeth, panic condition.
Notice the pregnant sows is ammonia poisoning.

Why is there such a high mortality in the piglets ?
1. Prenatal asphyxia can also be due to prenatal. In this case, the hogs are special acidosis in utero. At this point the piglets the blood pH from 6.0 to pH also may be affected. Due to the vitality of these pigs have no life.
2. the characteristic of the meconium birth of a pig?
3. you must have surfactant deficient piglets.
4. you see mulberry heart disease neonatal piglets?
5. do you have the villous atrophy in piglets?
Wrong thinking.
There is no shortage of animals symptom, not the problem that we do not do something in the feed mixture.
Solving the problem is that some of the material to be removed from the feed mixture!
Wisely.
This is a serious nutrition physiological causes. I feel sorry for the animals, and the economic damage is enormous.
I have been doing for years. There are a lot of practical experience.
It is not easy, but it can be removed.
Unfortunately my English is not good. Excuse Me.
Sincerely, Mester Károly, agricultural engineer, Hungary,
30-years pig feeding experience.
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02-Oct-2015Mome Nokubonga Competitive advantage of piggery

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