Page 2 of articles about PCV2 in Articles

This article deals with the factors to be considered for deciding the optimal time of vaccination against PCV2  and shows the results of a study that evaluates the efficacy of different vaccination programs in PCV2 subclinically infected piglets.

Recently, PCV2 has been suggested as a pathogen able to induce diarrhoea in pigs, and PCV2-enteric disease (PCV2-ED) as a separate entity within porcine circovirus diseases (PCVDs) has been proposed.

This article proves that, at least "in vitro" order matters. In PRRSV/PCV2 mixed infections, the consequences are worse when PRRSV infection occurs before or concurrently to PCV2 infection, than when the sequence is reversed.

The high seroprevalences found supports the feeling you get in the field that PCV2, PRRSV, and SIV are very widespread viruses in our farms.

Distinct PCV2 variants are circulating in commercial pig farms. How the vaccine induced immunity may affect PCV2 evolution?

The benefit on antibiotic use is particularly clear when herds are multicontaminated by pathogenic virus and bacteria, allowing also to maintain animal welfare and operational productivity, but a good immunity against PCV2 is effective also in high health status herds.

It is a test that allows the verification of the carrying out and/or the quality of the vaccination in cases such as the entrance of piglets for their fattening.

The first cause of PCV vaccine failure is the purchase and use of generic vaccine of low quality and titre, but the exact cause of the failures can occur in various forms.

The veterinarian is called from a new established herd in Brittany because some clinical signs appear on gilts from the two first deliveries introduced in the farm.

During 2012 PCV2 variants or mutants (mPCV2) appeared in the USA and were frequently associated with PCV-associated diseases. The possibility of lack of cross-protection of commercial vaccines against mPCV2 is now of major concern.

Reducing piglets mingling in farrowing and nursery facilities was found to delay the infectious process leading to a lower number of early infections. Clustering piglets by litter in small units after weaning also decreased significantly the probability of early infection.

What further increases profitability in pig production is not minimizing costs, but maximizing revenue.

Despite the evident usefulness of laboratory diagnosis, we must not forget that a field necropsy allows us to us recover part of the loss caused by the death of the animal.

PCV2 vaccination of the gilts prior to insemination with PCV2 spiked-semen showed a clear decrease of viral load in the gilts as well as a much lower viral excretion that were compatible with foetus protection.

The virus is highly resistant in the environment, showing also high resistance to chemical and thermal treatments.

Infection of pigs occurs at birth but maternal immunity suppresses viremic infection in growing pigs until immunity wanes around 10 weeks of age, when a hidden infection re-appears.