Page 3 of articles about PCV2 in Articles

Might be there is a little doubt nowadays on the beneficial clinical and economic effects of PCV2 vaccination in subclinical infections in piglets, but… which is the role that sows play in the whole picture?

This economic model was create to provide a decision support tool for farmers and veterinarians, who can enter their own data to calculate the severity of PMWS on their farm, the cost of disease and what could be the most cost-effective control measure.

One of the greatest long-term potentials for genomic selection is to identify specific genes or genomic regions with a significant impact on pig health and to aid selection for disease resistance and/or disease tolerance.

It is believed that TTSuVs themselves do not directly cause any disease but they could potentiate coinfecting pathogens and participate as triggering factors for diseases like PCVDs and PRDC.

PRRSV by itself may induce mild-to-severe respiratory disease but rarely results in mortality.

PCV2 may reach embryos/foetuses upon transplacental spread during viremia or upon insemination with contaminated semen.

It has been shown that the efficacy of PCV2 vaccines was dramatically decreased in the context of husbandry deviations such as excess of cross-fostering, mingling at weaning or very large groups in nursery.

One month after the beginning of the outbreak of reproductive failure in sows, acute respiratory disorder and diarrhoea that was non-responsive to antibiotics started in weaned pigs aged 6-8 weeks...

The efficacy of PCV2 vaccines to control PCV2 associated diseases has been clearly demonstrated not only in piglets but also in sows. Moreover, it is also feasible to vaccinate sows and their offspring without hampering its growth during the rearing period.

The prevalence of pleurisy is surprisingly high among pigs at slaughter. A recent review of available data ranged from 12.5% in the UK, 26% in Spain, to 41% of individual pigs slaughtered in one Norwegian study.

Experimental Mhyo and PCV2 co-inoculations have resulted in somewhat divergent results.

PCV2 can be shed into semen by infected boars and research suggests that the virus can then be transmitted to gilts and sows by AI causing reproductive failure but there is no evidence that PCV2 in semen has effects on various characteristics of sperm cells.

Genomic changes can be associated with increased virulence or decreased immunogenicity.

The PCV2 has been associated, in an ongoing way and throughout time, with more pathologies called, collectively, diseases associated with the PCV2 (PCVDs): PCV2 systemic disease, PCV2 lung disease, PCV2 enteric disease, PCV2 reproductive disease,PCV2 subclinical infection and porcine dermatitis and nephropaty syndrome.

Novelties about the excretion routes, the transmission routes, the risk factors, etc., and explanation about how the PCV2 becomes a pathogenic virus.

The most effective way of avoiding the production losses associated with the PCV2 consists in avoiding the appearance of the viraemia by inducing a passive or an active immunity that minimizes the circulation of the virus in the farm.