Page 5 of articles about PCV2

PCV2 vaccination of the gilts prior to insemination with PCV2 spiked-semen showed a clear decrease of viral load in the gilts as well as a much lower viral excretion that were compatible with foetus protection.

The virus is highly resistant in the environment, showing also high resistance to chemical and thermal treatments.

Infection of pigs occurs at birth but maternal immunity suppresses viremic infection in growing pigs until immunity wanes around 10 weeks of age, when a hidden infection re-appears.

Might be there is a little doubt nowadays on the beneficial clinical and economic effects of PCV2 vaccination in subclinical infections in piglets, but… which is the role that sows play in the whole picture?

This economic model was create to provide a decision support tool for farmers and veterinarians, who can enter their own data to calculate the severity of PMWS on their farm, the cost of disease and what could be the most cost-effective control measure.

One of the greatest long-term potentials for genomic selection is to identify specific genes or genomic regions with a significant impact on pig health and to aid selection for disease resistance and/or disease tolerance.

It is believed that TTSuVs themselves do not directly cause any disease but they could potentiate coinfecting pathogens and participate as triggering factors for diseases like PCVDs and PRDC.

PRRSV by itself may induce mild-to-severe respiratory disease but rarely results in mortality.

PCV2 may reach embryos/foetuses upon transplacental spread during viremia or upon insemination with contaminated semen.

It has been shown that the efficacy of PCV2 vaccines was dramatically decreased in the context of husbandry deviations such as excess of cross-fostering, mingling at weaning or very large groups in nursery.

One month after the beginning of the outbreak of reproductive failure in sows, acute respiratory disorder and diarrhoea that was non-responsive to antibiotics started in weaned pigs aged 6-8 weeks...